Euthyroid Hyperthyroxinemia – Euthyroid Conditions
Über den Vortrag
Der Vortrag „Euthyroid Hyperthyroxinemia – Euthyroid Conditions“ von Carlo Raj, MD ist Bestandteil des Kurses „M1 – Thread 2 – Bones-Neuro-Behavior“.
Quiz zum Vortrag
What causes euthyroid hyperthyroxinemia?
- Increased binding proteins
- Increased T4
- Increased conversion of T3 to T4, leading to decreased T3
- Decreased estrogen leads to decreased binding proteins
- Decreased albumin leads to increased TBG binding to T4
What increases glycosylation of thyroid-binding globulin (TBG), leading to decreased clearance?
- An increased concentration of estrogen
- Hepatitis
- Acute intermittent porphyria
- Amiodarone excess
- Methadone
Which medication does NOT increase the levels of thyroid-binding globulin (TBG)?
- Progesterone-only contraceptives
- Methadone
- Amiodarone
- Propanolol
- Tamoxifen
What is the pathophysiology of euthyroid hyperthyroxinemia in a pregnant woman?
- A high level of estrogen increases the level of thyroid-binding globulin (TBG), decreases free T4, and increases T4 production.
- Increased estrogen increases the level of TBG, decreasing bound T3.
- Delivery of the thyroid hormone to the fetus decreases the available thyroid hormone for the mother, leading to increased production.
- Increased TBG due to decreased bound T4 leads to a spike in estrogen until TBG levels even out.
- Decreased TBG due to increased delivery of T4 to the fetus leads to increased production of TBG.
What percentage of T4 is normally bound to thyroid-binding globulin (TBG) in the bloodstream?
- 70%
- 16%
- 99%
- 15%
- 80%
Dozent des Vortrages Euthyroid Hyperthyroxinemia – Euthyroid Conditions
Carlo Raj, MD
Dr. Carlo Raj is a Physician and Lecturer at Becker’s Healthcare, in Illinois, USA, and the CEO and founder of Indus Intellect Virtual MedEd, a medical education consulting company.
He obtained his MD from Medical University of the Americas (MUA) and is an international lecturer and public speaker.
Within Lecturio, Dr. Raj teaches courses on Pathology.
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